![]() ![]() We conclude that endogenous opioids inhibit the vasoconstriction of these vascular beds during fetal asphyxia. In contrast, kidney and carcass blood flows decreased, and their vascular resistances increased. Heart, brain, and adrenal blood flows increased due to the increase in blood pressure, with no changes in their vascular resistances. El rango de distribucin del pH sanguneo de la arteriaumbilical fue corregido en la clnica mediante la puntuacin del, obteniendo los valores de asfixia neonatal de. Asphyxia caused hypertension, and the fetal arterial blood pressure further increased when asphyxiated fetuses received naloxone. pH o de la BE de asfixia neonatal es un rango en lugar de un punto fijo. Naloxone solution or the solvent alone was added for the last 20 min. Fetal asphyxia was induced by reducing the uterine blood flow with an inflatable occluder around the common internal iliac artery to approximately 50% of control for 40 min. Thirteen fetal sheep (0.8-0.9 of gestation) were chronically catheterized. Este tipo de estrangulacin como hemos comentado se produce por un lazo que rodea el cuello. We examined the hypothesis that endogenous opioids modify the cardiac output distribution during asphyxia due to changes in the vascular resistance of some fetal organs. La estrangulacin es una forma de asfixia mecnica que se produce por la constriccin del cuello con las manos, con el antebrazo, con un lazo que rodea el cuello o con cualquier otra estructura que presiona sobre el cuello. Intravenous administration of the opioid receptor antagonist naloxone to asphyxiated fetal sheep increases the arterial blood pressure. ![]()
0 Comments
Leave a Reply. |
AuthorWrite something about yourself. No need to be fancy, just an overview. ArchivesCategories |